Obviously English zoo’s functioned as breeding places for animals containing agents of Spongiform Encephalopathy to transmit them into parts of Africa and into Oman and Saudi Arabia. “London zoo officials have written to other British zoos suggesting that ‘careful consideration’ should be given before animals at risk are exported to foreign zoos or for reintroduction programmes. London zoo’s circular says that any ungulate fed properietary meal between 1980 and 1989 may be affected.”
“There is growing interest in the reintroduction of captive-bred animals to the wild for conservation programmes and these reintroductions provide a route for the accidental transmission of infectious diseases into free-living populations…”.
In April 1989 an Arabian oryx cow died in Londons Regent’s Park because of BSE. Three month later, in July 1989, her nine month old calf, was shipped to Saudi Arabia. He died there on Aug 18, 1989. In Phoenix, Arizona, USA 216 Arabian oryx are awaiting to be shipped to Saudi Arabia.
In Oman the import of the BSE agents from Great Britain was kept hidden for four years. In Jan./Feb. 1989 two cows there showed clinical symptoms indicative of BSE. They had been exported from one affected herd in Great Britain in 1985. Their BSE-illnesses were the first cases to be diagnosed outside Great Britain. From January 1988 to March 1989 fully vaccinated children in Oman were attacked by an outbreak of paralytic poliomyelitis, indicative of immunosuppression. Soldiers from the USA, being active there at that time, became victims of “Gulf War Syndrome”, a disease indicative of retrovirus-particles connected with BSE. (See chapter 5)
In Serengeti Park in Tanzania in 1994 during 14 days over an 1,000 km distance 1,000 out of 3,000 lions died showing neurological symptoms reminding of Spongiform Encephalopathy. The lions are affected by the retrovirus Feline Immunodeficiency virus (see chapter 3.9). 95% of the young cheetahs there did not survive in 1994. 80% of the lions in Kruger National Park, South Africa, bordering to Mozambique, are infected by Feline Immunodeficiency Virus.
Jackals and foxes since 1978, wild dogs since 1991, uncounted hyaenas, foxes and leopards died in East Africa, showing neurological symptoms like the lions.
In 1994 50% of the buffalos and 25% of gnus in Serengeti died, the surviving are in a bad health condition.
Imported and domestic cows are dying in Uganda.
Dromedars in the north of Somalia are showing Bovine Spongiform Encephalopathy-symptoms.
Measles-viruses are in most of the cases named in articles in scientific papers to be responsible for those neurological and immunosuppressing diseases of seals, wales, dolphins, of elk, of buffalos, gnus, of lions, leopards, cheetahs, jaguars, jackals, hyaenas, and foxes. But they were never proven to be the causes for these mass-diseases. And they are unlikely to have mutated at so many places in the USA, England, Germany, Tanzania, Kenya and on coasts in North-America and Europe to cross the species barrier to such an extent and at so many places. For millions of years they had not affected f.e. cats. So we have to count for an immunosppressive agent enabling the infection of measles viruses.